Skip to main navigation Skip to search Skip to main content

A mathematical model of pHi regulation in central CO 2- Chemoreception

  • Stony Brook University

Research output: Chapter in Book/Report/Conference proceedingConference contributionpeer-review

Abstract

In CO2 chemosensitive neurons, an increase in CO2 (hypercapnia) leads to a maintained reduction in intracellular pH (pH i) while in non-chemosensitive neurons pHi recovery is observed. The precise mechanisms for the differential regulation of pH i recovery between these cell populations remain to be identified; however, studies have begun to explore the role of Na+/H+ exchange (NHE). Here we present the results of a mathematical model that explores pHi regulation in central CO2 chemoreception, and in particular the role of the isoform NHE-5 (present in brainstem neurons). We found that NHE-5 kinetics leads to pHi recovery in response to simulated hypercapnia as we already reported for the isoforms NHE-1 and NHE-3. Further, we wanted to test whether our model can reproduce common experimental protocols like NH4+ pre-pulse experiments designed to alter pHi.

Original languageEnglish
Title of host publication33rd Annual Northeast Bioengineering Conference - Engineering Innovations in Life Sciences and Healthcare, NEBC
PublisherInstitute of Electrical and Electronics Engineers Inc.
Pages300-301
Number of pages2
ISBN (Print)1424410339, 9781424410330
DOIs
StatePublished - 2007
Event33rd Annual Northeast Bioengineering Conference, NEBC - Stony Brook, NY, United States
Duration: Mar 10 2007Mar 11 2007

Publication series

NameProceedings of the IEEE Annual Northeast Bioengineering Conference, NEBEC
ISSN (Print)1071-121X

Conference

Conference33rd Annual Northeast Bioengineering Conference, NEBC
Country/TerritoryUnited States
CityStony Brook, NY
Period03/10/0703/11/07

Fingerprint

Dive into the research topics of 'A mathematical model of pHi regulation in central CO 2- Chemoreception'. Together they form a unique fingerprint.

Cite this