Abstract
We consider four mechanisms by which apparent discontinuities in the distribution of antisociality could arise: (1) executive genes or hormonal systems, (2) multiplicative interactions of predisposing factors, (3) environmental tracking into a limited number of social roles, and (4) cross-generational gene-environment interactions. A more explicit consideration of complex self-organizing dynamic systems may help us understand the maintenance of antisocial subpopulations.
| Original language | English |
|---|---|
| Pages (from-to) | 560-561 |
| Number of pages | 2 |
| Journal | Behavioral and Brain Sciences |
| Volume | 18 |
| Issue number | 3 |
| DOIs | |
| State | Published - 1995 |
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